Metabolic and endocrine disorders

Insulin resistance

Reversible in most cases, and the silent antechamber of type 2 diabetes. The earlier it is measured, the less it costs to correct.

Insulin resistance happens when your cells stop responding well to insulin and the pancreas compensates by producing more. For years fasting glucose can look normal while fasting insulin is already high. That is why many people find out late: they measured the thing that gives nothing away.

Nutritionally this is not about eliminating carbohydrates but about changing their type, amount and distribution through the day, and about recovering skeletal muscle mass, the tissue that disposes of most glucose. Measurement matters here: if you lose muscle while losing weight, insulin sensitivity gets worse even though the scale congratulates you.

  • Fasting insulin and HOMA-IR read alongside your body composition, not in isolation
  • Carbohydrate distribution built around your real routine, without extreme restriction
  • Priority on preserving and gaining skeletal muscle mass
  • Control of visceral fat, the depot most associated with the problem
  • Follow-up with bioimpedance every 8 to 12 weeks

This page is educational material from a registered dietitian, not a diagnostic tool. Diagnosis and medical treatment are your doctor’s remit; clinical nutrition accompanies that treatment, it does not replace it.

Frequently asked questions

No. It is an earlier stage. If it persists long enough and the pancreas tires, it becomes prediabetes and then type 2 diabetes. Caught early it responds well in a good proportion of cases, though how far and how fast depends on the person and on how long it has been developing.

Yes, and more often than people think. What matters is not weight but where the fat sits and how much muscle you carry. Someone at normal weight with low muscle mass and high visceral fat can be insulin resistant. That is exactly the profile an ordinary scale misses.

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